Advanced Search Results For "Glycation End Products, Advanced metabolism"
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Glycolaldehyde-derived advanced glycation end products promote macrophage proliferation via the JAK-STAT signaling pathway.
Publication Type: Academic Journal
Source(s): Molecular biology reports [Mol Biol Rep] 2023 Jul; Vol. 50 (7), pp. 5849-5858. Date of Electronic Publication: 2023 May 25.
Abstract: Background: Advanced glycation end products (AGEs) are heterogeneous proinflammatory molecules produced by a non-enzymatic glycation reaction between reducing sugars (and their metabolites) and biomolecules with amino groups, such as proteins. Although...
Receptor for the Advanced Glycation End Products ( RAGE ) Pathway in Adipose Tissue Metabolism.
Publication Type: Academic Journal
Source(s): International journal of molecular sciences [Int J Mol Sci] 2023 Jul 01; Vol. 24 (13). Date of Electronic Publication: 2023 Jul 01.
Abstract: Advanced glycation end products (AGEs) are mediators in the process of cellular dysfunction in response to hyperglycemia. Numerous data indicate that the accumulation of AGEs in the extracellular matrix plays a key role in the development of obesity-re...
Alpha cell receptor for advanced glycation end products associate with glucagon expression in type 1 diabetes.
Publication Type: Academic Journal
Source(s): Scientific reports [Sci Rep] 2023 Aug 09; Vol. 13 (1), pp. 12948. Date of Electronic Publication: 2023 Aug 09.
Abstract: Hypoglycemia in type 1 diabetes associates with changes in the pancreatic islet α cells, where the receptor for advanced glycation end products (RAGE) is highly expressed. This study compared islet RAGE expression in donors without diabetes, those at r...
Echinacoside-Zinc Nanomaterial Inhibits Skin Glycation by Suppressing the Transcriptional Activation of the Receptor for Advanced Glycation End-Products.
Publication Type: Academic Journal
Source(s): ACS nano [ACS Nano] 2023 Jul 25; Vol. 17 (14), pp. 14123-14135. Date of Electronic Publication: 2023 Jul 05.
Abstract: Glycation is a nonenzymatically catalyzed spontaneous reaction that eventually leads to the formation of advanced glycation end-products (AGEs), which can bind to the receptor for AGEs (RAGE). The consequences are oxidative damage, an inflammatory resp...
Dietary advanced glycation end products (dAGEs): An insight between modern diet and health.
Publication Type: Academic Journal
Source(s): Food chemistry [Food Chem] 2023 Jul 30; Vol. 415, pp. 135735. Date of Electronic Publication: 2023 Feb 24.
Abstract: Advanced glycation end products (AGEs) are formed by a series of chemical reactions of amino acids, peptides, proteins, and ketones at normal temperature or heated non-enzymatic conditions. A large amount of AGEs derived from Maillard Reaction (MR) dur...
Role of profilin-1 in vasculopathy induced by advanced glycation end products (AGEs).
Publication Type: Academic Journal
Source(s): Journal of diabetes and its complications [J Diabetes Complications] 2023 May; Vol. 37 (5), pp. 108415. Date of Electronic Publication: 2023 Feb 04.
Abstract: Aims: To construct a simple and feasible rat model to mimic diabetic vasculopathy by chronic injection of advanced glycation end products (AGEs) and further determine the role of profilin-1 in vasculopathy in AGE-injection rats.Methods: Sprague-Dawley ...
Evidence that methylglyoxal and receptor for advanced glycation end products are implicated in bladder dysfunction of obese diabetic ob / ob mice.
Publication Type: Academic Journal
Source(s): American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2023 Oct 01; Vol. 325 (4), pp. F436-F447. Date of Electronic Publication: 2023 Aug 10.
Abstract: Glycolytic overload in diabetes causes large accumulation of the highly reactive dicarbonyl compound methylglyoxal (MGO) and overproduction of advanced glycation end products (AGEs), which interact with their receptors (RAGE), leading to diabetes-assoc...
Effects of advanced glycation end products (AGEs) on the differentiation potential of primary stem cells: a systematic review.
Publication Type: Academic Journal
Source(s): Stem cell research & therapy [Stem Cell Res Ther] 2023 Apr 11; Vol. 14 (1), pp. 74. Date of Electronic Publication: 2023 Apr 11.
Abstract: The formation and accumulation of advanced glycation end products (AGEs) have been associated with aging and the development, or worsening, of many degenerative diseases, such as atherosclerosis, chronic kidney disease, and diabetes. AGEs can accumulat...
Receptor for Advanced Glycation End Products: Dementia and Cognitive Impairment.
Publication Type: Academic Journal
Source(s): Drug research [Drug Res (Stuttg)] 2023 Jun; Vol. 73 (5), pp. 247-250. Date of Electronic Publication: 2023 Mar 08.
Abstract: The pathophysiological processes of dementia and cognitive impairment are linked to advanced glycation end products (AGEs) and their receptor (RAGE).The neurofibrillary tangles (NFTs) of abnormally hyperphosphorylated tau protein and senile plaques (SP...
RAGE-TLR4 Crosstalk Is the Key Mechanism by Which High Glucose Enhances the Lipopolysaccharide-Induced Inflammatory Response in Primary Bovine Alveolar Macrophages.
Publication Type: Academic Journal
Source(s): International journal of molecular sciences [Int J Mol Sci] 2023 Apr 10; Vol. 24 (8). Date of Electronic Publication: 2023 Apr 10.
Abstract: The receptor of advanced glycation end products (RAGE) and Toll-like receptor 4 (TLR4) are important receptors for inflammatory responses induced by high glucose (HG) and lipopolysaccharide (LPS) and show crosstalk phenomena in inflammatory responses. ...